Green Tea Peripheral Neuropathy
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Matching Summaries of RecentPeer Reviewed Scientific Research Reports
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Match Strength: 13.485
The objective of the study was to investigate the subclinical visual deficit in type I and II diabetes, and its relationship with peripheral neuropathy. Thirty-two healthy volunteers, 20 patients with type I diabetes and 30 patients with type II diabetes were studied in a clinical neurophysiology setting. Luminance (VEPs) and chromatic visual evoked potentials (CVEPs) were recorded, with white-black, grey-black, red-green and blue-yellow sinusoidal gratings. The peak latencies of the VEP positive wave and CVEP negative wave were recorded. Ten patients with type I and 8 with type II diabetes ... Read More »
» Published in Neurol Sci. 2006 Nov;27(5):323-7. Comment in: Neurol Sci. 2006 Nov;27(5):299-300.
2. Peripheral nerve amyloidosis in sural nerve biopsies: a clinicopathologic analysis of 13 cases
Match Strength: 9.232
OBJECTIVE: Amyloidosis is a well-recognized but uncommon cause of peripheral neuropathy. Our objectives were to determine the overall prevalence of peripheral nerve amyloidosis in sural nerve biopsies and to evaluate the clinical and pathologic features of these lesions. METHODS: All available histologic and ultrastructural materials on biopsy tissue from 13 cases of peripheral nerve amyloidosis were examined. Muscle biopsies performed at the same time as the nerve biopsy were reviewed when available. Clinical data were collected on all patients. RESULTS: The prevalence of amyloidosis in sural ... Read More »
» Published in Arch Pathol Lab Med. 2000 Jan;124(1):114-8.
3. Green tea pain modulating effect in sciatic nerve chronic constriction injury rat model.
Match Strength: 8.054
We investigated the effect of green tea (GT) in unilateral chronic constriction injury (CCI) to the rat scaitic nerve. Five groups (n = 6 rats/group) sham group: rats which underwent operation but with no ligation to the scaitic nerve, and received tap water for two weeks before and for five weeks after the surgery. Four experimental groups underwent CCI to the right sciatic nerve, divided randomly as follows: group E were given tap water throughout the study. Group B received GT before and after CCI. Group C was given GT following CCI. Group D received GT for two weeks prior to CCI. Groups ... Read More »
» Published in Nutr Neurosci. 2006 Feb-Apr;9(1-2):41-7.
4. X-Linked inhibitor of apoptosis protein gene-based neuroprotection for the peripheral nervous system.
Match Strength: 6.930
OBJECTIVE: The recently discovered X-linked inhibitor of apoptosis protein (XIAP) is among the most potent inhibitors of programmed cell death. In the current experiment, we examine the potential of adenoviral XIAP gene delivery to protect neurons of the peripheral nervous system using in vitro models of amyotrophic lateral sclerosis (ALS) and diabetic neuropathy. METHODS: XIAP complementary deoxyribonucleic acid was fused in frame with the green fluorescent protein sequence and cloned into a first generation adenoviral vector. The impact of XIAP gene expression on glutamate-induced apoptosis ... Read More »
» Published in Neurosurgery. 2006 Jul;59(1):172-82; discussion 172-82.
5. Ocular Surface Changes with Applanation Contact Lens and Coupling Fluid Use After Argon Laser Photocoagulation in Noninsulin-Dependent Diabetes Mellitus
Match Strength: 6.833
PURPOSE: To describe the effect of coupling solutions used during laser photocoagulation on the ocular surface of patients with noninsulin-dependent diabetes mellitus (NIDDM). DESIGN: A prospective case-controlled study. METHODS: Ninety-two eyes of 46 NIDDM patients with clinically significant macular edema, poor metabolic control of diabetes, and peripheral neuropathy and 100 eyes of 50 normal control subjects were studied. The patients' eyes were assigned to argon green focal/grid laser photocoagulation using an applanation contact lens and one of the coupling fluids; 2% methocel, Thilo ... Read More »
» Published in Am J Ophthalmol. 2004 Sep;138(3):381-8. Comment in: Am J Ophthalmol. 2005 Apr;139(4):755; author reply 755-6., Am J Ophthalmol. 2005 Apr;139(4):757-8; author reply 758.
6. Myelin protein zero: mutations in the cytoplasmic domain interfere with its cellular trafficking.
Match Strength: 5.183
The cytoplasmic domain of myelin protein zero (MPZ), the principal protein of peripheral myelin, undergoes phosphorylation on several serine residues and a tyrosine group that is maximal during peak nerve myelination. Mutations that could affect MPZ phosphorylation cause the inherited neuropathy, Charcot-Marie-Tooth disease Type 1B. To investigate a possible role for phosphorylation in regulation of MPZ trafficking within the cell, we expressed wild-type and mutated MPZ-enhanced green fluorescent protein (GFP) fusion proteins in cultured Schwann-like cells. Whereas wild-type protein is present ... Read More »
» Published in J Neurosci Res. 2006 May 1;83(6):957-64.
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