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Barre-Lieou Syndrome
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1. Barre-Lieou syndrome and the problem of the obsolete eponym.
Match Strength: 32.255

Background: Eponym lists in major sources can give an aura of legitimacy to discredited diagnoses, as exemplified by the case of Barre-Lieou syndrome, a 'rare' vestibular disorder.Methods: A literature review for information on the posterior cervical syndrome of Barre-Lieou.Results: Barre-Lieou syndrome includes very common symptoms - tinnitus, dizziness, and head or neck pain - attributed to ischaemia caused by cervical sympathetic nerve compression. Its original description brings together many unrelated disorders, and its causative mechanism has been discredited. However, it appears ... Read More »
» Published in J Laryngol Otol. 2006 Oct 19;:1-4

2. Epidemiology of the Guillain-Barre syndrome.
Match Strength: 15.444

This review focuses on recent epidemiological findings on Guillain-Barre syndrome regarding incidence, antecedent events related to the disease, prognosis and prognostic indicators, and treatment. Moreover, this review summarizes recent observations on clinical variants of Guillain-Barre syndrome and their relationship with the prevailing clinical presentation of the disease. The epidemiological observations which have advanced the understanding of the pathogenesis of Guillain-Barre syndrome are also discussed. Publication Types: ... Read More »
» Published in Curr Opin Neurol. 2001 Oct;14(5):605-13.

3. Fisher/Gullain-Barre overlap syndrome in advanced AIDS.
Match Strength: 14.915

Human immunodeficiency virus-associated Guillain-Barre syndrome typically occurs early in HIV infection before AIDS develops. We report a 56-year-old male AIDS patient, who developed overlapping Fisher/Guillain-Barre syndrome despite markedly decreased CD4 cell counts. Serum anti-GQ1b IgG antibody was positive. His condition improved rapidly after immunoglobulin treatment. This indicates that Fisher/Guillain-Barre syndrome may occur even in severely immunosuppressed patients and that anti-ganglioside antibody production is possibly mediated by a T cell-independent process ... Read More »
» Published in J Neurol Sci. 2007 Jul 15;258(1-2):148-50. Epub 2007 Mar 27.

4. Early Guillain-Barre syndrome without inflammation
Match Strength: 14.608

A patient with typical acute Guillain-Barre syndrome died 72 hours after his first symptoms occurred, and an autopsy was performed 8 hours after his death. Extensive sampling of cranial and peripheral nerves, sensory ganglia, and autonomic nerves showed only minimal inflammatory lymphocytic and macrophage infiltrates. This case, one of the earliest studied extensively, represents an extreme example of a noninflammatory mechanism that has been proposed in some cases of Gullain-Barre syndrome ... Read More »
» Published in Arch Neurol. 1992 Sep;49(9):979-81.

5. The challenges of managing and treating Guillain-Barre syndrome during the acute phase.
Match Strength: 14.604

Guillain-Barre syndrome (GBS) is a randomly acquired inflammatory disease that affects approximately 2 persons in 100,000 annually. There have been no discriminating risk factors identified including age, sex, or race. The syndrome results in the demyelination of peripheral nerves, which leads to progressive motor weakness and paralysis. The critical care nurse should gain from this article an overview of Guillain-Barre syndrome during the acute phase. Included is the pathophysiology of the syndrome, clinical presentation, acute phase nursing assessment and management, and currently available ... Read More »
» Published in Dimens Crit Care Nurs. 2006 Nov-Dec;25(6):256-63.

6. Guillain-Barre syndrome and its treatment.
Match Strength: 14.401

Guillain-Barre syndrome typically presents with an acute ascending areflexic weakness, progressing over 4 weeks or less. The most common form of the disease is an acute inflammatory demyelinating polyneuropathy, but other forms with primarily axonal pathologies are well documented. The association of Guillain-Barre syndrome with a range of antecedent infections, particularly Campylobacter jejuni enteritis, is also established. A range of serological and neurophysiological investigations can assist in making an accurate diagnosis. Background information about the syndrome and the evidence base ... Read More »
» Published in Expert Rev Neurother. 2006 Oct;6(10):1569-74.

7. CSF tau protein: a new prognostic marker for Guillain-Barre syndrome.
Match Strength: 13.734

We measured the CSF tau protein levels in 26 patients with Guillain-Barre syndrome. The levels of the poor outcome group (Hughes grade at 6 months was between II and VI, n = 6) were higher than those of the good outcome group (0 or I, n = 20) (p < 0.0005). The higher levels of CSF tau may reflect axonal degeneration and could predict a poor clinical outcome in Guillain-Barre syndrome ... Read More »
» Published in Neurology. 2006 Oct 24;67(8):1470-2.

8. Guillain-Barre syndrome, greater Paris area.
Match Strength: 13.461

We studied 263 cases of Guillain-Barre syndrome from 1996 to 2001, 40% of which were associated with a known causative agent, mainly Campylobacter jejuni (22%) or cytomegalovirus (15%). The cases with no known agent (60%) peaked in winter, and half were preceded by respiratory infection, influenza-like syndrome, or gastrointestinal illness. Publication Types: Research Support, Non-U.S. Gov ... Read More »
» Published in Emerg Infect Dis. 2006 Jun;12(6):990-3.

9. A rare case of Guillain-Barre syndrome with pregnancy.
Match Strength: 13.420

Guillain-Barre syndrome is a rare neuorological disorder affecting 6-24/ 1,00,000 population during pregnancy. The case involved a 29-year-old woman conceived after 14 years of marriage presenting with 16 weeks pregnancy and clinical symptoms of Guillain-Barre syndrome. It was confirmed by nerve conduction test and patient was started on intravenous immunoglobulin. She had a rapid recovery following therapy and subsequent follow-up 3 months later showed complete recovery and healthy ongoing pregnancy. Various diagnostic and treatment options are discussed. Publication Types: Case ... Read More »
» Published in J Indian Med Assoc. 2006 May;104(5):269-70.

10. Anesthetic management of Guillain-Barre syndrome in pregnancy.
Match Strength: 13.243

We report the case of a 23-year-old woman who was diagnosed with an axonal type of Guillain-Barre syndrome at 16 weeks' gestation. The patient had severe motor loss but she was treated effectively with intravenous immunoglobulin, and she underwent cesarean delivery with epidural anesthesia at full term ... Read More »
» Published in J Clin Anesth. 2007 Jun;19(4):299-302.

11. Guillain-barre syndrome in an immunocompromised patient and coccidioidomycosis infection.
Match Strength: 13.089

A 70-year-old man was referred for evaluation of a 2-week history of numbness and progressive weakness in his lower and upper extremities and subsequently diagnosed with Guillain-Barre syndrome. The patient had been taking mycophenolate mofetil 500 mg twice daily and tacrolimus 6 mg daily for immunosuppression following a kidney transplant 2 years earlier. However, 5 weeks prior to presentation he had been diagnosed with pneumonia due to coccidioidomycosis and his tacrolimus dose was reduced to 1 mg daily to prevent a drug interaction with fluconazole, which was prescribed to treat the ... Read More »
» Published in Rev Neurol Dis. 2006 Spring;3(2):82-4.

12. Guillain-Barre syndrome--pathological connection with GvHD after allogeneic bone marrow transplantation.
Match Strength: 13.073

The Guillain-Barre syndrome (GBS) could be a manifestation of neurotoxicity caused by multiple factors due to allogeneic bone marrow transplantations (alloBMT). In this paper we present a case of a 40-year old woman with chronic myeloid leukemia after alloBMT from her HLA identical brother. She developed the second grade of acute hepatic graft versus host disease (GvHD) shortly after alloBMT followed by chronic form. We observed Guillain-Barre syndrome probably triggering by GvHD in this patient. Publication Types: Case ... Read More »
» Published in Ann Transplant. 2006;11(3):10-1.

13. Campylobacter, polyneuropathy, and Guillain-Barre syndrome in Denmark, 1994-2003.
Match Strength: 13.024

We examined the incidence of Campylobacter infections, Guillain-Barre syndrome and unspecified polyneuropathies in an ecological study from 1994 to 2003. The increase in Campylobacter cases in Denmark in the 1990s was comparable to an increase in cases of unspecified polyneuropathies; however, the incidence of Guillain-Barre syndrome cases remained stable during the period. Publication Types: Research Support, Non-U.S. Gov ... Read More »
» Published in Scand J Infect Dis. 2007;39(2):160-2.

14. Treatment of Guillain-Barre syndrome induced by cyclosporine in a lung transplant patient.
Match Strength: 12.899

We report the case of a 58-year-old man with severe chronic obstructive pulmonary disease who developed acute quadriparesis during the post-operative period following bilateral lung transplantation after receiving cyclosporine for immunosuppression. Electromyography with nerve conduction study and cerebrospinal fluid analysis supported a diagnosis of Guillain-Barre Syndrome, which improved upon the discontinuation of cyclosporine, replacement with tacrolimus, and initiation of plasmapheresis. We propose the discontinuation of cyclosporine and initiation of plasmapheresis as a treatment for ... Read More »
» Published in J Heart Lung Transplant. 2006 Jan;25(1):140-3. Epub 2005 Nov 14. Comment in: J Heart Lung Transplant. 2006 Nov;25(11):1387.

15. Guillain-Barre syndrome in northern China. Relationship to Campylobacter jejuni infection and anti-glycolipid antibodies.
Match Strength: 12.787

Guillain-Barre syndrome has been considered to be primarily an acute inflammatory demyelinating polyneuropathy (AIDP). Our experience with Guillain-Barre syndrome in northern China differs from the traditional concept. Electrophysiologically and pathologically, most of our patients have motor axonal degeneration with minimal cellular inflammation, which we have termed 'acute motor axonal neuropathy' (AMAN). The current studies were undertaken to characterize prospectively the clinical, electrophysiological, and serological features of Guillain-Barre syndrome, defined clinically, in northern ... Read More »
» Published in Brain. 1995 Jun;118 ( Pt 3):597-605.

16. Progress in Guillain-Barre syndrome.
Match Strength: 12.759

This editorial review summarizes and critically analyses reports published in the preceding 18 months on the pathogenesis of Guillain-Barre syndrome, with particular emphasis on the role of ganglioside antibodies, antecedent infections, and the concept of molecular mimicry. It concludes with an appraisal of currently available and proposed therapies. Publication Types: Editorial, ... Read More »
» Published in Curr Opin Neurol. 2001 Oct;14(5):597-604.

17. Guillain-Barre and Miller Fisher syndromes occurring with tumor necrosis factor alpha antagonist therapy.
Match Strength: 12.534

OBJECTIVE: Diverse neurologic syndromes have been described in association with tumor necrosis factor alpha (TNFalpha) antagonist therapy for inflammatory arthritides and Crohn's disease. The objective of this study was to review the occurrence and clinical features of Guillain-Barre syndrome and its variant, the Miller Fisher syndrome, during TNFalpha antagonist therapy. METHODS: The postmarketing database of the US Food and Drug Administration (FDA) was searched, following our experience with a patient with rheumatoid arthritis in whom the Miller Fisher syndrome variant of the Guillain-Barre ... Read More »
» Published in Arthritis Rheum. 2006 May;54(5):1429-34.

18. BiPAP in early guillain-barre syndrome may fail.
Match Strength: 12.445

BACKGROUND: Non-invasive mechanical ventilation (BiPAP) has been introduced for use in neuromuscular respiratory disease such as amyotrophic lateral sclerosis and myasthenia gravis. There is no experience in Guillain-Barre syndrome. METHODS: We describe for the first time the use of BiPAP to assist in the work of breathing in two consecutive patients with progressing Guillain-Barre syndrome (GBS) and marginal pulmonary function. RESULTS: Our initial attempts to use BiPAP in GBS and early neuromuscular respiratory failure were totally unsuccessful. There was marked initial improvement; however, ... Read More »
» Published in Can J Neurol Sci. 2006 Feb;33(1):105-6.

19. Magnetic resonance imaging of nerve root inflammation in the Guillain-Barre syndrome.
Match Strength: 12.380

We report gadolinium-enhancing nerve root lesions in a 52-year-old man with typical Guillain-Barre syndrome (GBS). This enhancement correlates well with the perineurial inflammatory and demyelinating processes known to characterize GBS and other inflammatory neuropathies. MRI should enable further exploration of patterns of disease in GBS and, with further study, perhaps assist in evaluating therapy ... Read More »
» Published in Neuroradiology. 1994;36(2):139-40.

20. Barium carbonate poisoning mimicking Guillain-Barre syndrome.
Match Strength: 12.366

Areflexic quadriplegia due to barium carbonate (rat poison) poisoning is described in two young patients. These cases very closely resembled Guillain-Barre syndrome. The various effects of barium carbonate along with the pathogenesis of hypokalaemic paralysis are highlighted. Publication Types: Case ... Read More »
» Published in J Assoc Physicians India. 2001 Jun;49:656-7.

21. Guillain-Barre syndrome following hepatitis B vaccination.
Match Strength: 12.341

A 52-year-old woman developed Guillain-Barre syndrome 10 weeks after immunization with recombinant hepatitis B vaccine. Common infectious causes of GBS were ruled out. The temporal relationship between GBS and hepatitis B virus (HBV) vaccination was suggestive of a vaccine-induced cause. The possible mechanisms of this very, rare complication are discussed. Publication Types: Case Reports, ... Read More »
» Published in Clin Exp Rheumatol. 2004 Nov-Dec;22(6):767-70.

22. Guillain-Barre syndrome: association with Campylobacter jejuni and Mycoplasma pneumoniae infections in India.
Match Strength: 12.184

BACKGROUND: Guillain-Barre syndrome is the most common cause of acute neuromuscular paralysis and is considered a post-infectious disease. METHODS: Twenty patients with Guillain-Barre syndrome admitted to the Neurosciences Centre at the All India Institute of Medical Sciences from November 1997 to August 1998 were investigated for evidence of antecedent infections. This case-control study included 2 controls for each patient, one a household control and the other an age- and sex-matched hospital control suffering from a neurological illness other than Guillain-Barre syndrome. Evidence of ... Read More »
» Published in Natl Med J India. 2006 May-Jun;19(3):137-9.

23. Facial diplegia with hyperreflexia-a mild Guillain-Barre Syndrome variant, to treat or not to treat?
Match Strength: 12.141

ABSTRACT: Guillain Barre Syndrome (GBS) is readily diagnosed when the presentation is that of ascending weakness and areflexia. Atypical presentations with preserved, and at times, brisk reflexes, can be a diagnostic dilemma. We describe a patient with GBS who presented with facial diplegia and hyperreflexia on examination and discuss management options ... Read More »
» Published in J Brachial Plex Peripher Nerve Inj. 2007 Apr 10;2:9.

24. Guillain-Barre syndrome complicated by myocarditis.
Match Strength: 12.096

Guillain-Barre syndrome (GBS) is an autoimmune disease affecting the peripheral nerves, and is frequently associated with triggering events several weeks prior to the onset of symptoms. We report the case of a 68-year-old female who was diagnosed with GBS and subsequently developed myocarditis. She was treated with inotropic support and intravenous immunoglobulin (IVIG), and her condition improved. This presentation of GBS complicated by myocarditis is very rare. We examined the literature regarding this association ... Read More »
» Published in Mt Sinai J Med. 2006 Dec;73(8):1126-8.

25. Landry-Guillaine-Barre syndrome as presentation of celiac disease.
Match Strength: 12.077

Celiac disease has been associated with a variety of neurological illnesses, most frequently cerebellar ataxia and peripheral neuropathy. We report presentation as Landry-Guillaine-Barre syndrome in a 28-year-old woman with previously unsuspected celiac disease. Publication Types: Case ... Read More »
» Published in Indian J Gastroenterol. 2007 Jan-Feb;26(1):42-3.

26. Magnetic resonance imaging of the cauda equina in Guillain-Barre syndrome.
Match Strength: 12.009

We report three patients (two children and one adult) with Guillain-Barre syndrome and magnetic resonance imaging evidence of gadolinium enhancement of the cauda equina and lumbar nerve roots. All three patients exhibited symmetric ascending paralysis and areflexia, and two (one child, one adult) suffered urinary incontinence and retention. Similar enhancement has been observed in patients with chronic inflammatory demyelinating polyneuropathy and suggests proximal nerve inflammation. Magnetic resonance imaging in Guillain-Barre syndrome and chronic inflammatory demyelinating polyneuropathy ... Read More »
» Published in Neurology. 1994 Jul;44(7):1334-6. Comment in: Neurology. 1995 May;45(5):1024-5.

27. Guillain-Barre syndrome after brachial plexus trauma: case report.
Match Strength: 11.988

The Guilllain-Barre syndrome (GBS) is an acute predominantly demyelinating polyneuropathy. In many cases GBS is preceding by infection, immunization, surgery or trauma. Although there are a few reports of GBS after head trauma, there is no report of this syndrome after brachial plexus injury. We report on a 51 years-old man who presented GBS fifteen days after a brachial plexus trauma. The polineuropathy resolved completely in a few weeks. We believe that GBS was triggered by the trauma that evoked an immune mediated disorder producing inflammation and demyelination of the peripheral nerves ... Read More »
» Published in Arq Neuropsiquiatr. 2006 Dec;64(4):1039-1040.

28. Cytomegalovirus disease with Guillain-Barre syndrome in a cadaver renal allograft recipient: cause or coincidence.
Match Strength: 11.949

Anecdotal reports of acute inflammatory demyelinating polyneuropathy (Guillain-Barre syndrome) with cytomegalovirus (CMV) suggested as the etiological agent have been described in transplant recipients with poor prognosis. We describe a 48-year-old man, a cadaveric renal allograft recipient on cyclosporine, mycophenolate mofetil and prednisolone, who developed febrile illness with unexplained anemia followed by progressive weakness of the upper and lower limbs. He was diagnosed as a case of Guillain-Barre syndrome (GBS). His CMV serology was positive by polymerase chain reaction (PCR). We ... Read More »
» Published in Int Urol Nephrol. 2007 Apr 21;

29. A Case of Chronic Demyelinating Polyneuropathy Resembling the Guillain-Barre Syndrome
Match Strength: 11.925

A case of demyelinating polyneuropathy is reported which was clinically and pathologically indistinguishable from the Guillain-Barre syndrome, with the exception that the symptoms progressed steadily for 7 months. Pathologically, the peripheral nerves and intraspinal roots showed selective demyelination with axonal sparing and patchy mononuclear inflammation. The posterior columns of the spinal cord also showed selective demyelination with axonal sparing. The simultaneous destruction of central and peripheral myelin is discussed in relation to the immune theory for the Guillain-Barre syndrome. ... Read More »
» Published in J Neurol Sci. 1977 May;32(1):45-52.

30. Urinary retention and sympathetic sphincter obstruction in axonal Guillain-Barre syndrome.
Match Strength: 11.914

A 62-year-old woman with axonal Guillain-Barre syndrome developed weakness and urinary retention simultaneously. The retention failed to recover for 10 months even after she regained the ability to walk. The patient exhibited no postural hypotension. Videourodynamics showed that the retention was caused not by the bladder paralysis but rather by internal (sympathetic) sphincter obstruction, which is extremely uncommon in peripheral neuropathies. We started the patient on an alpha-adrenergic antagonist, urapidil, at 30 mg/day, and this led to successful relaxation of the urethra and reduced the ... Read More »
» Published in Muscle Nerve. 2007 Jan;35(1):111-5.

31. Circulating transforming growth factor beta 1 (TGF-beta1) in Guillain-Barre syndrome: decreased concentrations in the early course and increase with motor function.
Match Strength: 11.900

OBJECTIVE: To delineate the possible implication of the immunosuppressive cytokine transforming growth factor beta 1 (TGF-beta1) in the pathogenesis of Guillain-Barre syndrome. Guillain-Barre syndrome is a disorder that may implicate cytokines in its pathogenesis. TGF-beta1 is a potent anti-inflammatory cytokine occasionally shown to be regulated in the course of demyelinating disorders. METHODS: The study measured circulating proinflammatory and anti-inflammatory cytokines from the progressing phase to early recovery in patients with Guillain-Barre syndrome. Plasma concentrations of TNF-alpha ... Read More »
» Published in J Neurol Neurosurg Psychiatry. 1998 Feb;64(2):162-5. Comment in: J Neurol Neurosurg Psychiatry. 1998 Feb;64(2):148.

32. Pathogenicity of Anti-Ganglioside Antibodies in the Guillain-Barre Syndrome
Match Strength: 11.752

Guillain-Barre syndrome (GBS) is a postinfectious inflammatory polyradiculo-neuropathy characterized by flaccid paralysis. Antibodies directed against glycolipid structures (gangliosides), which are highly expressed in the peripheral nervous system, are frequently detected in sera from GBS patients. These antibodies interfere with nerve conduction and have been shown to activate phagocytes via IgG receptors (FcgammaR). These findings support an important role of glycolipid-specific antibodies in the pathogenesis of GBS ... Read More »
» Published in Autoimmun Rev. 2004 Feb;3(2):61-8.

33. Vincristine-induced acute neurotoxicity versus Guillain-Barre syndrome: a diagnostic dilemma.
Match Strength: 11.645

We report the case of a patient with acute lymphoblastic leukaemia who, after the initiation of treatment with vincristine (VCR), developed a fulminant motor polyradiculoneuropathy resembling an axonal variant of Guillain-Barre syndrome (GBS). This report shows that differentiating between axonal GBS and VCR-induced acute neurotoxicity may be a challenge for clinicians ... Read More »
» Published in Eur J Neurol. 2007 Jul;14(7):826-8.

34. Case report: Hepatitis A preceding Guillain-Barre syndrome.
Match Strength: 11.638

A case of acute hepatitis A with Guillain-Barre Syndrome subtype AMAN (acute motor axonal neuropathy) in a 17-year-old male is reported. Serum and cerebrospinal fluid were positive for anti-hepatitis A virus (HAV) IgM, IgG, and IgA. The onset of the syndrome was evident in week 3 of illness. The remarkably high titers of serum anti-HAV IgG appeared unique to a hepatitis A patient with the syndrome. Phylogenetic analysis of the HAV genome detected in the serum and feces revealed genotype IIIA, circulating commonly in Pune, western India. 2006 Wiley-Liss, Inc. Publication Types: Case ... Read More »
» Published in J Med Virol. 2006 Aug;78(8):1011-4.

35. Guillain-Barre syndrome as a cause of reversible cardiomyopathy.
Match Strength: 11.597

Although autonomic dysfunction is a common manifestation of Guillain-Barre syndrome, cardiovascular involvement in this setting has rarely been reported in the literature. We describe a case of reversible left ventricular systolic dysfunction in a 60-year-old man with Guillain-Barre syndrome. Our patient had no history or signs of cardiac dysfunction on initial presentation. During the clinical manifestation of his autonomic dysfunction, he developed electrocardiographic changes accompanied by mildly elevated cardiac enzymes and severe left ventricular systolic dysfunction and segmental wall ... Read More »
» Published in Tex Heart Inst J. 2006;33(1):57-9.

36. GQ1b Antibody Testing in Guillain-Barre Syndrome and Variants.
Match Strength: 11.564

Guillain-Barre syndrome (GBS) is characterized by an ascending muscle paralysis with progressive loss of muscle stretch reflexes. Annually, approximately 2.4 cases per 100,000 population of GBS are reported. Variant forms do exist. These include the Fisher syndrome, GBS with ophthalmoplegia, Bickerstaff's brainstem encephalitis (BBE), and acute ophthalmoparesis without ataxia. In the last 15 years, attention has been directed towards the association of the GQ1b IgG antibody and several GBS variants, particularly the Fisher syndrome and those associated with ophthalmoparesis.We present three ... Read More »
» Published in Semin Ophthalmol. 2006 Oct-Dec;21(4):223-7.

37. Anti-GT1a IgG antibodies in a child with severe Guillain-Barre syndrome.
Match Strength: 11.507

This report describes a male, age 8 years 10 months, with severe Guillain-Barre syndrome after Campylobacter jejuni infection. The patient developed fulminant muscle weakness, external ophthalmoplegia, bulbar palsy, and respiratory distress. A high level of serum monospecific anti-GT1a immunoglobulin G antibody was detected. He was treated with intravenous immunoglobulins and artificial ventilation. Two years after the onset, the patient still suffered from residual leg weakness and foot drop. After 3 years and clinical recovery, the antibody was no longer detectable. This report presents the ... Read More »
» Published in Pediatr Neurol. 2006 Oct;35(4):277-9.

38. Postoperative delayed respiratory failure caused by Guillain-Barre syndrome--a case report.
Match Strength: 11.483

Postoperative respiratory failure caused by Guillain-Barre syndrome (GBS) is a rare complication after general anesthesia. We report a GBS patient who after receiving an operation for polycystic liver disease under general anesthesia developed weakness of upper and lower extremities on the 3rd postoperative day, with decreased deep tendon reflex, which ultimately evolved into respiratory failure. Slurred speech and bilateral ptosis were also noted. All these manifested an acute peripheral polyneuropathy combined with bulbar involvement. According to the clinical picture, CSF examination and ... Read More »
» Published in Acta Anaesthesiol Taiwan. 2007 Mar;45(1):43-6.

39. Guillain-Barre syndrome-like presentation in borderline leprosy with type-2 reaction.
Match Strength: 11.453

A 46-year-old man with borderline lepromatous leprosy with type-2 reaction being treated with multi-bacilliary-multiple drug therapy and steroids presented with an acute onset of flaccid quadriparesis. A nerve conduction study and CSF analysis were similar to that seen in Guillain Barre syndrome. Muscle weakness improved considerably with an increased dose of corticosteroid; after 6 months the patient recovered completely. Publication Types: Case Reports, ... Read More »
» Published in Dermatol Online J. 2006 Feb 28;12(2):21.

40. Plasmapheresis treatment in Guillain-Barre syndrome: potential benefit over IVIg in patients with axonal involvement.
Match Strength: 11.396

Response to therapeutic plasma exchange (TPE) was evaluated in patients diagnosed with Guillain-Barre Syndrome (GBS). Our aim was to assess response to TPE in patients who had failed treatment with intravenous immune globulin (IVIg). We conducted a retrospective chart review of 10 patients with the diagnosis of Guillain-Barre Syndrome who required TPE. Patients were identified by reviewing data from log books for TPE at The University of Connecticut Health Center, Farmington CT, USA. Patients who had failed IVIg treatment prior to being referred for TPE were also identified. Eight out of 10 ... Read More »
» Published in Ther Apher Dial. 2004 Oct;8(5):409-12.

41. Guillain-Barre syndrome occurring after adjuvant chemo-radiotherapy for endometrial cancer.
Match Strength: 11.245

BACKGROUND: The association between Guillain-Barre syndrome (GBS) and malignancy is uncommon and has not been previously reported in gynecological cancers. CASE: Our case documents this syndrome occurring in a patient shortly after completion of adjuvant chemo-radiotherapy for endometrial carcinoma. We review the current literature and discuss potential pathogenic mechanisms of this likely paraneoplastic association. CONCLUSION: GBS in cancer patients is a potentially life-threatening condition and should be differentiated from simple chemotherapy toxicity, particularly as effective treatment ... Read More »
» Published in Gynecol Oncol. 2006 Mar;100(3):615-7. Epub 2005 Oct 20.

42. Significance of phrenic nerve electrophysiological abnormalities in Guillain-Barre syndrome.
Match Strength: 11.232

The authors investigated whether the amplitude and latency of diaphragm compound muscle action potential helped predict respiratory failure in Guillain-Barre syndrome. Both variables were significantly but weakly correlated with vital capacity (VC) and were similar in unventilated (n = 60) and ventilated (n = 10) patients. In ventilated patients, motor loss severity, progression, and VC reduction were significantly greater, and bulbar dysfunction was more common. Predicting respiratory failure must rely on clinical features and VC ... Read More »
» Published in Neurology. 2005 Nov 22;65(10):1646-9. Comment in: Neurology. 2006 Jun 27;66(12):1961; author reply 1961.

43. Diagnostic Considerations in Guillain-Barre Syndrome
Match Strength: 11.165

Guillain-Barre syndrome (GBS) is a recognized entity for which the basis for diagnosis is descriptive in our present state of knowledge. Diagnosis rests upon pattern recognition of the clinical picture plus other features including elevated cerebrospinal fluid protein level, electrophysiological changes of marked slowing of conduction velocities, prolonged distal latencies, dispersion of the evoked responses, and frequent evidence of conduction block, together with pathological changes, when known, of low grade-inflammation and demyelination-remyelination in peripheral nerve. The precise ... Read More »
» Published in Ann Neurol. 1981;9 Suppl:1-5.

44. Atypical presentation of Guillain-Barre syndrome with acute hydrocephalus.
Match Strength: 11.121

A 32-year-old man was treated with a ventriculo-peritoneal shunt for acute panventricular hydrocephalus. On the third postoperative day, an ascending tetraparesis and subsequent respiratory failure developed. Based on the clinical findings, cerebrospinal fluid study and electrophysiological results, a diagnosis of Guillain-Barre syndrome (GBS) was made. Despite plasmaphoresis and immunoglobulin infusion, the patient was ventilator dependent and bed-bound for 4 months. One year later, he was able to walk without assistance and had only slightly wasting of the intrinsic hand muscles. This is the ... Read More »
» Published in Acta Neurochir (Wien). 2008 Jan;150(1):87-8; discussion 88. Epub 2007 Nov 12.

45. Bartonella henselae infection associated with Guillain-Barre syndrome.
Match Strength: 11.069

This is the first report of Guillain-Barre syndrome (GBS) related to Bartonella henselae infection. A 10-year-old girl had difficulty walking and marked myalgia. The search for all causes known to trigger GBS was negative. She was treated with intravenous immunoglobulins and recovered. Because she lived in a rural area and had a history of kitten contact, a specific serology for B. henselae infection was performed and confirmed an ongoing infection. She did not show any clinical typical feature of cat-scratch disease. B. henselae infection should be considered in the wide etiologic spectrum of ... Read More »
» Published in Pediatr Infect Dis J. 2006 Jan;25(1):90-1.

46. Elevated anti-sulfatide antibodies in Guillain-Barre syndrome in T cell depleted at end-stage AIDS.
Match Strength: 11.016

A 38-year-old man developed the Guillain-Barre syndrome (GBS) associated with untreated end-stage AIDS and CD4+ lymphocyte count of 3 cells/mm(3). The patient had serum high titer anti-sulfatide antibodies and responded well to infusion of immunoglobulin. The data suggest that elevated levels of anti-sulfatide antibodies may play a role in the pathogenesis of GBS in this patient, although a direct neurotropic effect of HIV virus cannot be excluded ... Read More »
» Published in J Neuroimmunol. 2007 Jun 27;

47. CSF neurofilament levels: a potential prognostic marker in Guillain-Barre syndrome.
Match Strength: 10.998

Long-term morbidity from Guillain-Barre syndrome (GBS) is caused by axonal damage. This prospective study demonstrated that neurofilaments (NfHs), a biomarker for axonal damage, were of prognostic value in GBS. CSF NfH levels correlated with the F score and Medical Research Council summed score and were higher in patients with neurophysiologic evidence of axonal degeneration compared to those without. Pathologically high CSF NfH levels (>0.73 ng/mL) predicted worse motor and functional outcome. Publication Types: Comparative ... Read More »
» Published in Neurology. 2006 Sep 26;67(6):1071-3.

48. Coronary artery bypass surgery in Guillain Barre syndrome.
Match Strength: 10.976

Guillain Barre Syndrome (GBS) is a rare autoimmune inflammatory polyneuropathy with established acute phase morbidity and mortality. Despite the positive outcome in majority of cases, there is hesitance in subjecting these patients to major surgical interventions under general anaesthesia. This case documents the successful undertaking of major coronary artery grafting under cardiopulmonary bypass and general anaesthesia, in a GBS patient. A review of the pathology is presented and the controversy addressed. Publication Types: Case ... Read More »
» Published in J Pak Med Assoc. 2006 Feb;56(2):88-9.

49. Inflammatory neuropathies.
Match Strength: 10.976

We discuss two of the most common of the acquired inflammatory neuropathies: Guillain-Barre syndrome and chronic inflammatory demyelinating polyneuropathy, as well as their variants. We review their clinical presentation, electrophysiologic findings, and management, highlighting knowledge gained from the recent literature. Unfortunately, although treatments exist for both Guillain-Barre syndrome and chronic inflammatory demyelinating polyneuropathy, none are completely curative and all have significant potential side effects and/or expense. Better understanding of the underlying ... Read More »
» Published in Curr Neurol Neurosci Rep. 2005 Feb;5(1):66-71.

50. Guillain-Barre syndrome triggered by influenza vaccination in a recipient of liver transplant on FK506.
Match Strength: 10.929

Guillain-Barre syndrome (GBS) has been rarely reported after liver transplantation and generally has good outcome. We report a liver transplant patient on FK506 (tacrolimus) who developed GBS 6 months after liver transplantation. There was no evidence of liver rejection or active infection. Despite treatment with intravenous immunoglobulin, the patient expired. GBS occurred despite downregulation of T cells by FK506, suggesting that humoral dysfunction might be the predominant mechanism of GBS in this report. Publication Types: Case ... Read More »
» Published in Liver Transpl. 2006 Oct;12(10):1537-9.

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